The hallmark of Standard Process is its glandular supplements, specifically the PROTOMORPHOGENS (PMG). Many health practitioners note that there is ABSOLUTELY NO SUBSTITUTE for the protomorphogens manufactured by Standard Process. The publication PROTOMORPHOLOGY - THE PRINCIPLES OF CELL AUTO-REGULATION was presented by Royal Lee and William Hanson in 1947. The TWO PRIMARY factors In the theory are that through alimentary ingestion (7) increased amounts of protomorphogen is supplied thereby assisting In a speedier recovery of tissue, and, (2) increased protomorphogen material acts 10 speed elimination of increased Natural Tissue Antibodies (NTA) in the blood. Although the complete text and theory is complex, the empirical evidence of protomorphogen efficacy remains unquestionable. They WORK! The following is a portion of a concise and condensed explanation of how PROTOMORPHOGENS work described by Royal Lee in 1956.

A protomorphogen (PMG) is that component of the cell

chromosome that is responsible for morphogenic determination of cell characteristics. It is the smallest unit of the cell blueprint assembly. It is the smallest unit of the gene system that guides the cell into its hereditary form as it grows, develops or repairs itself.

Without sufficient protomorphogen in its chromatin, the cell degenerates, de-differentiates, becomes senile and dies. The protomorphogen level in the cell is regulated by the fact that, while normally more is constantly being created by the cell nucleus, his antigenic and promotes the formation of antibodies (in the mammalian organism), which in turn control the levels of extracellular protomorphogen in blood and lymph.

Throughout the years, we have periodically pioneered remarkable "firsts". These "firsts" have not all been immediately accepted by the consensus of opinion. No method of measuring the effectiveness of protomorphogens by laboratory means has been discovered, just as was the case for many years with vitamins. The clinical response, however, can be demonstrated in a matter of minutes by an instrument such as the Endocardiograph.

Cytotrophic Extracts are manufactured under U.S. Patent #2,374,219 which states the "purpose of this patent is to provide an improved method of producing a sterilized dry substance from a juice." This sterilization takes place below pasteurization temperature of a juice, thus the synergistic agents, such as amino acids and enzymes are not destroyed. Cytotrophic Extracts are not drugs. They are composed of the mineral fractions of animal tissue which is found associated in the protein molecule. Nutritionally this would be considered in the category of meat extracts. Since hormones are not contained in these products, there are no contraindications as to their use, therefore they are sold as experimental FOOD PREPARATIONS. Naturally, no food products are subject to, or restricted by the Experimental Drug Law.

...and in another paper:

It may be assumed that the specific growth factors (the cellular blueprints known as protomorphogen (PMG) that are constantly being secreted by each cell into its surrounding fluids) are prevented from traveling very far by the influence of specific antibodies, known as Natural Tissue Antibodies (NTA). They must be destroyed, if allowed to build up in any concentration, they would promote cell growth and mitosis. Only if any specific organ becomes subject to overwork and consequent inflammation in some degree does this occur. (A kidney doubles in size in six months after its partner has been removed.) (Muscles grow if sufficient demand is made on their ability.)

Where disease has damaged an organ, such as tuberculosis in the cases of lung, or where the heart has hypertrophied by overwork, the ingestion of heart or lung PMG, as the case may be, may at first create adverse reactions of a toxic nature (malaise, tiredness), apparently by reason of the immediate proteolytic destruction of the ingested PMG by antibodies in the blood stream, that are present in higher amounts than normally, by reason of the long-standing inflammation of the specific organ.

But cardiographic recordings will show that within a few minutes after ingestion of the cardiac PMG the heart action changes for the better. It is hard to explain this reaction other than by assuming that the excess heart tissue antibody in the circulating blood has been reduced by combination with the ingested heart PMG. This is probably done without danger of stimulating the formation of more heart tissue antibody, since alimentary ingestion normally does not permit proteins to act as antigens. Parenteral introduction of such materials is another matter.

Other factors that assist in controlling NTA are allantoin, betaine, (probably be a depolymerizing effect), and the hormones of the gonads, thyroid, thymus, and adrenal. Thymus acts by promoting colloidal dispersion that physiologically opposes cortisone, which flocculates antigen into particulate dimensions that permit their ingestion by phagocytes (and then antibody formation). The thymus during the development age, prevents this and keeps PMG available for growth stimulation and ultimate enzyme digestion and renal elimination.

Thyroid hormone splits PMG off the chromatin reserves of the cell, or from absorbed stores in connective tissue. That is why thyroxin accelerated tadpole metamorphosis. It is also the reason why thyroxin increases the metabolic rate. The released PMG stimulates cell activities.